商业文章代写：为什么要测量心率
每分钟心脏节律性收缩的平均次数称为心率。静息心率通常在每分钟60到100次之间。心电图是一种有效的工具，用来推断异常心率和找出潜在的条件。心电图中显著的u波是窦性心动过缓的明确指标(Opie, 2004)。这是一个潜在的联系，表明血流动力学妥协。本例慢性窦性心动过缓病情严重，可能是造成患者全身乏力的原因。异丙肾上腺素已用于这种情况。在心电图中，起搏器的位置很容易找到。心电图上可见房室间隔。有效的起搏器通常是在起搏刺激高峰之后发现的，随后是窦性心律中的QRS波复合体(Ellestad, 2003)。由此可以了解正常和异常起搏器。平均动脉压来自心脏的收缩压和舒张压。它被计算为心输出量(CO)和全身血管阻力之间的平均值。
商业文章代写：为什么要测量心率
它通常在70 -100汞的愤怒(Ellestad, 2003)。地图值必须在平均范围内(Ellestad, 2003)。这意味着身体的重要器官接受的氧气比需要的少。降低患者MAP值可导致缺氧或心脏性Ishmeia。异丙肾上腺素作用的活性部位位于小动脉内膜介质内。具体来说，它们模拟了心脏平滑肌中β- a1和β- a2肾上腺素能受体的作用(Markel等，1993)。这一行动的总体效果是β2受体的血管舒张造成影响和改变意味着系统性动脉压。异丙肾上腺素类药物在心脏中起着类似交感神经的作用(Markel et al.， 1993)。也就是说，它们在交感神经系统中发挥激动剂的作用，而交感神经系统模仿儿茶酚胺的作用机制(Markel et al.， 1993)。
商业文章代写：为什么要测量心率
The average number of rhythmic contraction of the heart per minute is known as heart rate. Resting heart rate is usually between 60 to 100 beats per minute. ECG is an effective tool used to deduce abnormal heart rates and find out underlying conditions. Prominent u waves in ECG are definitive indicator for Sinus Bradycardia (Opie, 2004). This is an underlying association that indicates haemodynamic compromise. This chronic case of Sinus Bradycardia that has been severe could be the reason for the patient’s general fatigue and tiredness. Isoproterenol has been administered for this condition. In an ECG the pacemaker sites can be easily found. Atrial and ventricular spacing can be found on the ECG. Effective pacemaker is usually found subsequent to the pacing stimulus spike that is followed by the QRS complex in the sinus rhythm (Ellestad, 2003). From this the normal and abnormal pacemaker can be comprehended. Mean arterial pressure is derived from systolic and diastolic blood pressure in the heart. It is calculated as an average between cardiac output (CO) and systemic vascular resistance.
商业文章代写：为什么要测量心率
It is usually in the rage of 70 -100 Hg (Ellestad, 2003). It is imperative for MAP values to be within the average range (Ellestad, 2003). It means that the vital organs of the body are receiving lesser than required oxygen. Lowering of the MAP values in the patient could lead to hypoxia or Cardiac Ishmeia. The active sites which isoproterenol acts are inside tunica media of arterioles. Specifically they simulate the action of beta1- and beta2-adrenergic receptors in the smooth muscles of the heart (Markel et al., 1993). Overall effect of this action is that β2 receptor’s vasodilation causes impact and changes the mean systemic arterial pressure.Isoproterenol drug acts as a sympathomimetic in the heart (Markel et al., 1993). That is they function as an agonist found in the sympathetic nervous system that mimics the mechanism of action of catecholamine (Markel et al., 1993).